Histone H3.3K27M Represses p16 to Accelerate Gliomagenesis in a Murine Model of DIPG – Research at the Nicholas School
  
RESEARCH @ NICHOLAS | from discoveries to solutions
OUR FOCUS AREAS
OUR LABS & CENTERS
OUR FOCUS AREAS: OCEANS
OUR FOCUS AREAS: TECHNOLOGY
OUR FOCUS AREAS: ENVIRONMENTAL HEALTH
OUR FOCUS AREAS: ENERGY
OUR FOCUS AREAS: WATER
OUR FOCUS AREAS: ECOLOGY & CONSERVATION
OUR FOCUS AREAS: SUSTAINABILITY
OUR FOCUS AREAS: CLIMATE CHANGE
OUR FOCUS AREAS: ECONOMICS, POLICY & GOVERNANCE
OUR FOCUS AREAS: TOXICOLOGY
OUR FOCUS AREAS: WETLANDS
OUR FOCUS AREAS: FORESTS
OUR FOCUS AREAS: GEOSCIENCES
OUR FOCUS AREAS: ATMOSPHERIC SCIENCE
OUR FOCUS AREAS: FOOD SYSTEMS
OUR FOCUS AREAS: URBAN ENVIRONMENT

Histone H3.3K27M Represses p16 to Accelerate Gliomagenesis in a Murine Model of DIPG

Cordero, FJ, Huang, Z, Grenier, C, He, X, Hu, G, McLendon, RE, Murphy, SK, Hashizume, R, and Becher, OJ. Molecular cancer research : MCR (May 18, 2017).
Full text >

Nicholas faculty author(s):

view all recent publications >